By James D. Crapo, Jeffrey L. Glassroth, Joel B. Karlinsky, Talmadge E. King
Since its first ebook in 1965, this article has been the definitive medical publication within the box. Written through greater than a hundred overseas specialists, the focal point is at the scientific points of treating pulmonary affliction. The textual content is evidence-based and entire of sensible examples. Chapters are hugely established to supply easy access to info. The 7th version has been condensed right into a unmarried concise but accomplished volume.
A Brandon-Hill advised title.
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Extra resources for Baum's Textbook of Pulmonary Diseases
The absence of an injurious response to normal lung particle burdens appears to be the result, in part, of the unique role of resident lung defense cells, such as alveolar macrophages. Each alveolus contains an average of 12 macrophages, which are thought to process all particles reaching this region under normal conditions. Alveolar macrophages have been shown to have a blunted capacity for antigen presentation and mitogen production compared with other monocytic phagocytes, and thus they are able to process inhaled particles without stimulating excessive immunologic responses or lung inflammation.
The firing of Ib neurons, as of Ia neurons, occurs primarily in inspiration but, in contrast to the discharge of Ia neurons, is augmented by inputs from pulmonary stretch receptors. In animals, the discharge peak of I b is not as sharp and the decline in activity during expiration is slower than for Ia. In the absence of excitatory input from vagal stretch receptors, however, the discharge patterns of Ia and Ib neurons are similar. Like Ia neurons, some of the Ib neurons project to the spinal cord.
Type II cells are identified by their distinctive microvilli. In this micrograph, the overlying surfactant layer was removed by fixatives. The shape of alveoli in vivo approximates a smooth partial circle. Smoothing of the folds on the alveolar surface is accomplished by folding of alveolar septal membranes into the capillaries and by filling of tissue depressions with alveolar lining fluid containing surfactant at its surface. Changes in alveolar size are thought to occur primarily by folding and unfolding of the alveolar pleats, and this process minimizes stress tension on alveolar septal cells.
Baum's Textbook of Pulmonary Diseases by James D. Crapo, Jeffrey L. Glassroth, Joel B. Karlinsky, Talmadge E. King